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Table 16 also provides the agonists or agents used to induce vasoconstriction (preconstrict the arteries prior to inducing vasodilation), the receptor types targeted, the associated intracellular signaling mechanisms, and the expected vascular responses linked to each agonist, with a particular focus on endothelial-mediated effects, except for sodium nitroprusside (which is primarily used to assess endothelium-independent relaxation or to distinguish endothelial from VSMC contributions to vasodilation)

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Individuals with diabetes, active acute infection, and malignant tumors were excluded from the study